In the late winter of 1917, a handful of Indo-Chinese laborers crossing the US on their way to France stopped in Haskell County, Kansas for at least three days. They had limited contact with any local Americans, and what contacts there were, were casual at most. On the morning of 11 March 1918, Albert Martin Gitchell reported to sick call at Camp Funston, Kansas (on the site of the modern Fort Reilly). He complained of a high fever, aches and pains, and a cough. Usually, this would have meant isolation in a sick ward (which was done), but Gitchell was a cook, who had been serving food as late as the night before. By noon there were 107 influenza patients; in a week, over 500. By April, over 1,000. Even this would have been unremarkable if 46 hadn’t died–horribly, coughing lung matter out in their final moments, blue-in-the-face.
What no one appreciated just then was that this H1N5 strain of influenza (so-called for the proteins in the outer shell of the virus) might have started as early as 1916 in Britain or France–to this day it is unclear. There were no centralized reporting mechanisms then, no CDC or WHO that anyone could recognize as such. Modern researchers believe that this strain of influenza may have been a close genetic match to the 1898 influenza, a milder form that swept the globe starting in China (as the flu always does) from October 1898 to March of 1899–flu season. It seems likely that the Vietnamese laborers carried the virulent Asian bug into Kansas, where it crossed with another strain, though the truth is unknowable.
What was remarkable about it wasn’t the “knock-me-down fever” that the flu was called, but that so many (proportionately) died, and not the elderly, infirm or very young who were usually flu fatalities. These were young people, healthy and in a prime state. Four deaths would have been odd, but 46 such horrible deaths
At the time, medicine was in a state of transition. The only widely accepted vaccine was for smallpox; there were no antibiotics; there were still physicians whose medical training took about four months and did not involve looking at a cadaver. This bug spread from town to town, state to state, country to country. By June it had spread to most of the ports of debarkation and exploded worldwide. The Wilson administration was aware of the pandemic but forbade widespread news coverage of it because it would have been bad for morale. The British and French, Italians and everyone else had similar reasons for not covering it as the bodies stacked up in the morgues, ships arrived in port with bloody flux all over the decks and dead in the hundreds. For this reason, the only major European power that covered this plague in their mass media–the newspapers–was Spain, and that’s how it came to be known as the Spanish Flu.
This flu hit the sufferers suddenly and often violently. Caregivers came to know which sufferers were going to survive and which would not within the first few hours the symptoms presented. Extreme sufferers (about 20%) turned blue, cracked their ribs coughing, spewed black fluids from their mouth and nose, and died in hours..sometimes minutes. There was no treatment save codeine for their cough, and it hit those between the ages of 20 and 40 the hardest. Post-mortems showed the extreme sufferers were drowned in the detritus of their own immune systems that attacked the invading virus so vigorously that it killed their hosts. In milder forms, the affected simply weakened and died (40%) within days. The mildly afflicted–the lucky or strong 40%–suffered from a malaise that often lasted for years, sometimes for the rest of their lives.
By the second week in November 1918–when the War to End All Wars was ending–leading clinicians in the US and Britain, Russia (where the Revolution came to a brief halt) and even Japan were calculating the end of the human race. Most gave humanity perhaps six months to live. Many believed it had to be a new plague…a resurrected, reconstituted Black Death.
The American Army’s fatality roles 1917-18 were doubled by influenza. Large cities like Detroit and Chicago, Paris and London monitored traffic and imposed quarantines; rural communities and isolated islands stopped traffic altogether, frequently at gunpoint. A streetcar in Johannesburg loaded with passengers and departed a stop and five blocks later unloaded all of the 21 passengers and the conductor–dead. Children deprived of their caregivers starved to death, especially in urban areas. Funeral directors ran out of coffins and embalming fluids, which combined with shortages of gravediggers resulted in mass cremations: one in Vienna, Austria was said to have contained over 10,000 dead. Entire North African and Chinese villages were burned. Actuaries in the United States dropped the average life expectancy for 1918 from 55 to 37.
By the end of November, the rate of infection slowed, and by the end of January 1919, it became clear that the crisis had passed. It came again that winter, and once more in the winter of 1920-21, but the virulence seemed decreased, and the number of fatalities far less. Nearly 100 million people worldwide were killed directly or indirectly by the 1918 influenza; one in four (about a billion) were affected one way or another–sickened and survived like my mother’s father, overworked and weakened like my father’s father, or watched whole populations wiped out like my father’s uncle. In closing:
- There are no lab samples, despite years of searching in graves: thus, there are no specific vaccines against the 1918 influenza.
- Since the 1918 bug struck those in the prime of life–those who make vaccines–it is not clear that one could be made available if it should strike again.
- The failure rate of the annual flu vaccine is about 30%; in bad years, like 2017-18, it rises as high as 60%. However, even failed or non-specific vaccines decrease the symptoms and the likelihood of retransmission.
- Herd immunity is best sustained when 92% or more of any given population has been vaccinated, even with a non-specific vaccine.
- The “reaction” to the flu shot shows that it is not only working but that the sufferer has already been exposed and is likely contagious.
- About 20% of adults do not get regular flu shots.
Got your flu shot yet? Why not?
Thursday is Pi Day–3.14. It started out in 1988 as a celebration of mathematics by Larry Shaw of the San Francisco Exploratorium. The US Congress passed a non-binding resolution in 2009 recognizing 14 March as Pi Day. Nominally, this clever holiday has been celebrated or observed by throwing pies, holding mathematical symposiums, eating pizza and other more or less benign activities.
However, like many other things, Pi Day has been hijacked by…other interests. In 2005, an Oregon State physics major named Bobby Henderson sent an open letter to the Kansas State Board of Education, which was then struggling with creationism and intelligent design requirements alongside more scientifically accepted versions of Earth’s origins. He suggested that it is as likely that a Flying Spaghetti Monster created everything as it was any other deity. The most significant phrase reads:
I don’t have a problem with religion. What I have a problem with is religion posing as science. If there is a god and he’s intelligent, then I would guess he has a sense of humor.Bobby Henderson
Not exactly 97 Theses nailed to a door, but in the 21st century, it was enough. Soon, the Church of the Flying Spaghetti Monster–the Pastafarians–was born. A book entitled The Gospel of the Flying Spaghetti Monster was released in 2006. There are websites, and more books, and the odd, odd convention, and somehow piracy and other odd things got tossed in the chaotic mix. Mostly the Pastafarians are polking fun at organized religion, especially when it pretends to circumvent falsifiability.
Oh, and Pi Day is celebrated by some Pastafarian sects as recognition of a related deity. It is observed by reverently eating pizza…at least, according to my late buddy Bill, may Pasta